mouse tissue sections (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc
mouse tissue sections
Mouse Tissue Sections, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 91 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/brain+tissue+sections/Mouse+Brain+Tissue+Control+Extracts/pm41634038-264-5-27
Average 94 stars, based on 91 article reviews
Mouse Tissue Sections, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 91 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/brain+tissue+sections/Mouse+Brain+Tissue+Control+Extracts/pm41634038-264-5-27
Average 94 stars, based on 91 article reviews
mouse tissue sections - by Bioz Stars,
2026-10
94/100 stars
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other:Article Title: Harnessing macrophage-drug conjugates for allogeneic cell-based therapy of solid tumors via the TRAIN mechanism. Article Snippet: Postincubation, cells were gently detached using Accutase (Sigma-Aldrich, A6964), stained with DRAQ7 dead cell stain (BioLegend, 424001) according to the manufacturer’s instructions, and resuspended in flow cytometry buffer for analysis using a BD FACSCanto II flow cytometer. Article Title: Harnessing macrophage-drug conjugates for allogeneic cell-based therapy of solid tumors via the TRAIN mechanism Article Snippet: Membranes were incubated overnight at 4 °C with primary antibodies: anti-clathrin heavy chain (1:1000; Abcam, ab21679), anti-TfR1 (1:1000; Cell Signaling Technology, 13113), anti-CD81 (D3N2D) (1:1000; Cell Signaling Technology, 56039), anti-CD9 (D8O1A) (1:1000; Cell Signaling Technology, 13174), anti-flotillin-1 (D2V7J) XP (1:1000; Cell Signaling Technology, 18634), anti-Alix (E6P9B) (1:1000; Cell Signaling Technology, 92880), anti-MSR1 (1:1000, Cell Signaling Technology, 17275), anti-GM130 (D6B1) XP (1:1000; Cell Signaling Technology, 12480), anti-Annexin V (1:1000; Cell Signaling Technology, 8555), anti-ICAM1 (E3Q9N) XP (1:1000; Cell Signaling Technology, 67836), anti-GAPDH (1:5000, Invitrogen, PA5-85074), anti-lamin B1 (1:1000, Abcam, ab16048), anti-β-actin (1:10000; Proteintech, 66009-1) or anti-α-tubulin (DM1A) (1:1000; Cell Signaling Technology, 3873), diluted in 2.5% w/v non-fat dry milk in TTBS. Incubation:Article Title: Involvement of IL-17 in Secondary Brain Injury After a Traumatic Brain Injury in Rats. Article Snippet: The pro-inflammatory activity of interleukin 17, which is produced by the IL-23/IL-17 axis, has been associated with the pathogenesis of traumatic brain injury (TBI).. The study investigated the potential role of IL-17 in secondary brain injury of TBI in a rat model. Our data showed that the levels of IL-17 increased from 6 h to 7 days and peaked at 3 days, in both the CNS and serum, which were consistent with the severity of secondary brain injury.. The IL-23 inhibitor suberoylanilide hydroxamic acid (SAHA) treatment markedly decreased the expressions of IL-17 and apoptosis-associated proteins cleaved caspase-3 and increased the protein ratio of Bcl-2 (B cell lymphoma/leukemia-2)/Bax (Bcl-2-associated X protein). Marker:Article Title: Involvement of IL-17 in Secondary Brain Injury After a Traumatic Brain Injury in Rats. Article Snippet: The pro-inflammatory activity of interleukin 17, which is produced by the IL-23/IL-17 axis, has been associated with the pathogenesis of traumatic brain injury (TBI).. The study investigated the potential role of IL-17 in secondary brain injury of TBI in a rat model. Our data showed that the levels of IL-17 increased from 6 h to 7 days and peaked at 3 days, in both the CNS and serum, which were consistent with the severity of secondary brain injury.. The IL-23 inhibitor suberoylanilide hydroxamic acid (SAHA) treatment markedly decreased the expressions of IL-17 and apoptosis-associated proteins cleaved caspase-3 and increased the protein ratio of Bcl-2 (B cell lymphoma/leukemia-2)/Bax (Bcl-2-associated X protein). |
